Scientists solve mystery of how Zika virus targets key immune cell. New finding shed light on how to stop virus from spreading.
ROCKVILLE, MD – The Zika outbreak of 2015 and 2016 is having lasting impacts on children whose mothers became infected with the virus while they were pregnant.
Although viruses such as herpes simplex can infect the eye’s cornea and Zika virus has been found in corneal tissue and tears, new research from Washington University School of Medicine in St. Louis suggests the cornea can resist infection from the novel coronavirus.
Zika virus is capable of replicating and spreading infectious particles within the outermost cells lining the vaginal tract, according to new research.
Researchers at Washington University School of Medicine in St. Louis have found that the strain of Zika that circulated in Brazil during the microcephaly epidemic that began in 2015 was particularly damaging to the developing brain.
Two different UC San Diego research teams identified the same molecule — αvβ5 integrin — as Zika virus’ key to brain cell entry. They found ways to take advantage of the integrin to both block Zika virus from infecting cells and turn it into something good: a way to shrink brain cancer stem cells.
Researchers in China have discovered how a Zika virus protein reshapes its host cell to aid viral replication. The study, which will be published December 23 in the Journal of Cell Biology, reveals that the viral protein NS1 converts an interior cellular compartment called the endoplasmic reticulum (ER) into a protective region where the virus can survive and replicate. Blocking this process could be a novel therapeutic strategy to treat patients infected with Zika or similar viral pathogens, such as the yellow fever and dengue viruses.
GAINESVILLE, Fla. — Feed mosquitoes more nitrogen when they’re young, and the adults are less likely to transmit the Zika virus, University of Florida scientists say. Now, researchers want to know why, and they’re determined to discover how the findings…